Congenital self-healing reticulohistiocytosis (also known as “Hashimoto–Pritzker disease,” and “Hashimoto–Pritzker syndrome”) is a condition that is a. -Hashimoto-Pritzker disease, or congenital self-healing reticulohistiocytosis, was initially described in neonates, or during the first months of life, as a cutaneous. The diagnosis of congenital self-healing reticulohistiocytosis (Hashimoto–Pritzker syndrome) was considered based upon histopathogical findings along with.

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The real incidence of congenital self-healing reticulohistiocytosis CSHR may be underreported because of its high rate of spontaneous resolution and lack of clinical recognition.

Currently, there prifzker no criteria other than clinical that can reliably distinguish CSHR from cutaneous involvement by disseminated Langerhans cell histiocytosis LCH.

We found that no significant difference was seen in the histologic features and the expression of E-cadherin, Ki, and PHH3 between the two groups; thus supporting the theory that CSHR and LCH represent different ends of a spectrum of the same condition.


Congenital self-healing reticulohistiocytosis

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Orphanet: Hashimoto Pritzker syndrome

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Material and methods Histologic evaluation. Reprints not available from the authors.